In vitro, VLDL, IDL, and HDL stimulate distinct procoagulant activities in human mononuclear cells, while LDL suppresses these effects, suggesting a complex regulatory role of lipoproteins in thrombosis.
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VLDL/IDL/HDL may enhance while LDL suppresses procoagulant activities; leaves open whether these in vitro effects influence clinical thrombosis risk.
Schwartz et al. (1981) studied this question.
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