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October 1, 1998British Journal of PharmacologyOpen Access

Mechanism of block by tedisamil of transient outward current in human ventricular subepicardial myocytes

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Authors

EWErich WettwerWest German Heart and Vascular Center EssenHHHerbert M. HimmelBayer (Germany)GAGregory AmosThe University of Queensland

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Implication

Does not support clinical use of tedisamil; leaves open its I(to) effects for further in vivo study.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Wettwer et al. (1998) studied this question.

synapsesocial.com/papers/6a73dc63e3ef2a9a940dd74ahttps://doi.org/10.1038/sj.bjp.0702110
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Tedisamil inactivates transient outward K+ current in rat ventricular myocytes1989 · 82 citations
  2. 2Transient outward current in human and rat ventricular myocytes1993 · 133 citations
  3. 34-Aminopyridine and the early outward current of sheep cardiac Purkinje fibers.1979 · 257 citations
  4. 4Transient outward and delayed rectifier currents in canine atrium: properties and role of isolation methods1996 · 156 citations
  5. 5Ionic Mechanism of Action Potential Prolongation in Ventricular Myocytes From Dogs With Pacing-Induced Heart Failure1996 · 576 citations