Intrinsic ultradian fluctuations of plasma catecholamines do not appear to control cardiovascular parameters during recumbency, suggesting morning increases in blood pressure and heart rate are driven by direct sympathetic neural input in response to posture and activity.
No takes yet. Share an insight, caveat, or question.
Does not support catecholamine fluctuations as drivers of recumbent cardiovascular parameters; leaves open direct neural input for postural blood pressure and heart rate rises.
Schöfl et al. (1997) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: