Population
Baby-hamster kidney cells infected with foot-and-mouth-disease virus
Design
Preclinical
Authors
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Challenges guanidine mechanism hypothesis in FMDV; leaves open later-cycle targets for further mechanistic studies.
Higher concentrations of guanidine can inhibit foot-and-mouth-disease virus replication later in the growth cycle, challenging the hypothesis that it acts specifically on viral RNA polymerase formation.
Black et al. (1969) studied this question.
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