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April 1, 1989Journal of Hypertension

A major structural abnormality in the renin gene of the spontaneously hypertensive rat

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Authors

Nilesh J. Samani
Nilesh J. SamaniGeneral CardiologyWBWilliam J. BrammarGoogle (United States)JSJ. D. SwalesGeneral / Preventive / Lipids

Discussion

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Implication

No immediate clinical implications from this rat model; leaves open whether renin intron variants influence human hypertension.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Samani et al. (1989) studied this question.

synapsesocial.com/papers/6a83dd1cfd07e211f9fe1fbahttps://doi.org/10.1097/00004872-198904000-00002

Topics

Hypertension management
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1MUTATIONS IN THE FIRST INTRON OF THE SHR RENIN GENE DISRUPT PUTATIVE REGULATORY ELEMENTS1995 · 10 citations
  2. 2Nucleotide Variations in Intron 1 of the Renin Gene of the Spontaneously Hypertensive Rat1998 · 10 citations
  3. 3Polymorphisms Of The Renin Gene Promoter In Spontaneously Hypertensive And Wistar‐Kyoto Rats2001 · 6 citations
  4. 4Structural alterations of the renin gene in stroke-prone spontaneously hypertensive rats: examination of genotype-phenotype correlations1990 · 65 citations
  5. 5A NEW RESTRICTION FRAGMENT LENGTH POLYMORPHISM IN THE FIRST INTRON OF THE SPONTANEOUSLY HYPERTENSIVE RAT RENIN GENE1994 · 6 citations