Why the study?
Does aspirin ingestion or specific bleeding disorders alter platelet adhesion and thrombus formation on the subendothelium?
Does aspirin ingestion or specific bleeding disorders alter platelet adhesion and thrombus formation on the subendothelium?
Aspirin prevents platelet thrombus formation without impairing initial subendothelial adhesion, providing early mechanistic rationale for its use as an antithrombotic agent.
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Distinguishes aspirin's selective blockade of thrombus formation from adhesion in vitro; leaves open translation to clinical antithrombotic benefit.
Weiss et al. (1975) studied this question.
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