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August 1, 1992Journal of VirologyOpen Access

The interaction of two groups of murine genes determines the persistence of Theiler's virus in the central nervous system

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Authors

JBJean‐François BureauCentre National de la Recherche Scientifique XMXavier MontagutelliInstitut Pasteur SLS. LefebvreUniversity of Groningen

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Implication

Mouse strain differences in TMEV persistence map to two loci; leaves open relevance to human demyelinating disease susceptibility.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Bureau et al. (1992) studied this question.

synapsesocial.com/papers/6a8af4e73c68152a7ce9505b https://doi.org/10.1128/jvi.66.8.4698-4704.1992

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Major histocompatibility complex-conferred resistance to Theiler's virus-induced demyelinating disease is inherited as a dominant trait in B10 congenic mice1990 · 52 citations
2Immunoglobulins and complement in demyelination induced in mice by Theiler's virus.1988 · 35 citations
3Persistent infection of oligodendrocytes in Theiler's virus‐induced encephalomyelitis1983 · 244 citations
4Class II-restricted T cell responses in Theiler’s murine encephalomyelitis virus-induced demyelinating disease. IV. Identification of an immunodominant T cell determinant on the N-terminal end of the VP2 capsid protein in susceptible SJL/J mice1991 · 47 citations
5Partial suppression of Theiler's virus‐induced demyelination in vivo by administration of monoclonal antibodies to immune‐response gene products (Ia antigens)1986 · 102 citations