Key result
Intravenous or direct injection of serotonin into the pulmonary circulation results in an increase in pulmonary vascular resistance caused by vasoconstriction.
Design
Review
Authors
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Hypothesis-generating for serotonergic pathways in acute pulmonary embolism; prospective validation required before any clinical consideration.
The text reviews historical evidence regarding the role of serotonin in causing bronchoconstriction and pulmonary hypertension following pulmonary embolism.
Pat O. Daily (1966) conducted a review in Pulmonary Embolism. Serotonin was evaluated. Intravenous or direct injection of serotonin into the pulmonary circulation results in an increase in pulmonary vascular resistance caused by vasoconstriction.
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