Key result
Hepatocyte growth factor (HGF) and its c-Met receptor are upregulated in post-MI rat hearts, and HGF delays the death of cultured cardiac myocytes exposed to oxidative stress.
Why the study?
Does hepatocyte growth factor attenuate myocyte death due to oxidative stress in acute MI?
Does hepatocyte growth factor attenuate myocyte death due to oxidative stress in acute MI?
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Hepatocyte growth factor may provide a novel endogenous cardioprotective mechanism to attenuate myocyte death and maladaptive hypertrophy following myocardial infarction.
Ian Dixon (2001) conducted an editorial in Myocardial infarction. Hepatocyte growth factor (HGF) was evaluated. Hepatocyte growth factor (HGF) and its c-Met receptor are upregulated in post-MI rat hearts, and HGF delays the death of cultured cardiac myocytes exposed to oxidative stress.
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