Why the study?
The pathophysiology and deleterious cardiac effects of Access Based Cardiac Disease in end stage kidney disease patients on dialysis remain poorly understood, especially in the hyper-acute phase.
Case Report (n=1)
No
This review highlights the pathophysiology of hyper-acute Access Based Cardiac Disease in end-stage kidney disease patients on dialysis.
Answers are grounded in this paper and cite their sources.
Raises awareness of hyper-acute access-related cardiac effects in dialysis patients; leaves open whether targeted interventions alter outcomes.
Fifty years after the creation of the first arteriovenous access, little is known about the deleterious cardiac effects of this pathological circuit. Understanding the pathophysiology of Access Based Cardiac Disease (ABCD) is critical, given the fact that cardiac abnormalities are the leading cause of morbidity and mortality in end stage kidney disease patients on dialysis. However, this has been a challenge, as many of hemodialysis patients have preexisting cardiac disease, they invariably have volume overload from salt and water retention, and many heart failure symptoms can be masked by regular efficient dialysis. ABCD can be hyper-acute (minutes to hours), acute (days to weeks), or chronic (months to years). In this manuscript, authors will focus on the hyper-acute form.
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A et al. (2019) conducted a case report in Access Based Cardiac Disease (n=1). Arteriovenous loop graft creation was evaluated on Hemodynamic stability.
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