Key result
The combination of late potentials on signal-averaged ECG and non-dipolar QRST area map identified patients with sustained ventricular tachycardia after myocardial infarction with 92% sensitivity and 60% specificity.
Why the study?
Reentrant mechanisms involving local conduction delay and increased refractory heterogeneity are important in sustained ventricular tachycardia during the chronic phase of myocardial infarction, but noninvasive detection methods require evaluation.
Does the combination of signal-averaged electrocardiogram and body surface QRST area map accurately differentiate patients with ventricular tachycardia in the chronic phase of myocardial infarction?
Cross-Sectional (n=67)
Yes
Does the combination of signal-averaged electrocardiogram and body surface QRST area map accurately differentiate patients with ventricular tachycardia in the chronic phase of myocardial infarction?
Absolute Event Rate: 91.7% vs 40%
p-value: p=<0.05
Noninvasive detection of conduction delay and repolarization abnormality using signal-averaged ECG and QRST area maps is useful for identifying patients with ventricular tachycardia in the chronic phase of myocardial infarction.
May support noninvasive post-MI VT risk stratification; leaves open prospective validation before clinical use.
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Tsunakawa et al. (1992) conducted a cross-sectional in Chronic myocardial infarction with or without sustained ventricular tachycardia (n=67). Sustained ventricular tachycardia vs. No ventricular tachycardia was evaluated on Presence of late potentials on signal-averaged ECG or non-dipolar QRST area map (p=<0.05). The combination of late potentials on signal-averaged ECG and non-dipolar QRST area map identified patients with sustained ventricular tachycardia after myocardial infarction with 92% sensitivity and 60% specificity.
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