Key result
Intensive insulin therapy may reduce free fatty acid toxicity in the ischemic heart by inhibiting lipolysis and preventing excessive fatty acid entrance into mitochondria.
Design
Editorial
Authors
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This editorial suggests that the beneficial effects of glucose-insulin-potassium therapy in acute myocardial infarction may be mediated by reducing free fatty acid toxicity in the ischemic heart.
This editorial suggests that the beneficial effects of glucose-insulin-potassium therapy in acute myocardial infarction may be mediated by reducing free fatty acid toxicity in the ischemic heart.
Christian A. Seiler (1998) conducted a letter in acute myocardial infarction. glucose-insulin-potassium therapy was evaluated. Intensive insulin therapy may reduce free fatty acid toxicity in the ischemic heart by inhibiting lipolysis and preventing excessive fatty acid entrance into mitochondria.
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