Why the study?
The association between the angiotensin-converting enzyme (ACE) gene deletion/insertion (D/I) polymorphism and myocardial infarction or other cardiac pathologies has been controversial, with conflicting findings across studies.
Case-Control (n=214)
Observes higher ACE D allele frequency in acute myocardial infarction, suggesting a possible link between the DD genotype and left ventricular hypertrophy.
OBJECTIVE: Since the initial report of the association of the deletion/insertion (D/I) polymorphism in the gene for angiotensin-converting enzyme (ACE) with myocardial infarction (MI), there has been considerable controversy. Some have found the D allele to be associated with MI, coronary heart disease (CHD) or other cardiac pathologies, while others have not. In view of the clinical importance of the ACE as a major marker of cardiovascular diseases, we investigated the I/D polymorphism of the ACE gene in Turkish patients with acute myocardial infarction in comparison with control subjects. METHODS AND RESULTS: Polymerase chain reaction, and agarose gel electrophoresis techniques were used to determine the ACE genotype in 214 subjects. The frequencies of ACE D and ACE I allele among the patients with acute myocardial infarction were 65.54% and 36.45% and in the control subjects 57.62% and 42.37%, respectively. ACE DD genotypes were found higher in patients with left ventricular hypertrophy (LVH) than without LVH (55.6% vs. 37.7%; X2: 2.534, p > 0.05). CONCLUSIONS: The ACE D allele is more frequent in patients with acute myocardial infarction than in controls. Moreover ACE DD genotype might be associated with an increased risk of left ventricular hypertrophy.
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Karaali et al. (2004) conducted a case-control in Acute myocardial infarction and left ventricular hypertrophy (n=214). ACE I/D gene polymorphism (D allele / DD genotype) vs. Control subjects / patients without left ventricular hypertrophy was evaluated on ACE D allele frequency in acute myocardial infarction vs controls, and ACE DD genotype in patients with vs without left ventricular hypertrophy (p=>0.05).
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