Why the study?
This article systematically analyzes the impact of chronic psychoemotional and physiological stress on cardiovascular hemodynamics and neuroendocrine regulation, including mechanisms of sympathoadrenal and hypothalamic-pituitary-adrenal axis hyperactivation, cortisol hypersecretion, resting tachycardia, reduced heart rate variability, endothelial dysfunction, and sustained arterial hypertension.
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This review discusses the pathophysiological mechanisms by which chronic psychoemotional and physiological stress leads to neuroendocrine hyperactivation, reduced heart rate variability, endothelial dysfunction, and sustained arterial hypertension.
Maqolada surunkali psixoemotsional va fiziologik stressning yurak-qon tomir tizimi gemodinamikasi hamda neyroendokrin boshqaruviga ta'siri tizimli tahlil qilinadi. Simpatoadrenal tizim hamda gipotalamo-gipofizar-buyrakusti bezi (GGB) o'qining uzluksiz giperfaollashuvi, kortizol gipersekretsiyasi, tinch holatdagi taxikardiya, yurak ritmi variabelligining (YRV) pasayishi, endotelial disfunksiya va barqaror arterial gipertenziya shakllanish mexanizmlari yoritiladi.
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Maxbuba Kaxorova (2026) conducted a review in Cardiovascular dysregulation and hypertension associated with chronic stress. Chronic psychoemotional and physiological stress was evaluated on Cardiovascular hemodynamics and neuroendocrine regulation (cortisol, heart rate variability, endothelial function, and arterial blood pressure).
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