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July 21, 2025

Supplementary Figure S6 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer

Ya-Yun Cheng Beth L. Worley Zaineb Javed Amal T. Elhaw Priscilla W. Tang Sarah Al-Saad Shriya Kamlapurkar Sierra R. White Apoorva Uboveja Karthikeyan Mythreye Katherine M. Aird Traci A. Czyzyk Nadine Hempel

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Key Points

Loss of MPZL3 expression is associated with increased epithelial-mesenchymal transition (EMT) in ovarian cancer.
Higher immune cell infiltration correlates with lower MPZL3 levels, indicating its influence on the tumor microenvironment.
The study utilized cell adhesion assays to examine the relationship between MPZL3 and immune cells.
Understanding MPZL3's role may lead to new insights in ovarian cancer treatment strategies.

Implication

Analysis shows MPZL3 expression influences immune cell infiltration in ovarian cancer, suggesting a role in EMT.

Abstract

Supplementary Figure S6. Association between MPZL3 expression and immune cell infiltration.

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Cite This Study

Cheng et al. (2025) studied this question.

synapsesocial.com/papers/689a060ee6551bb0af8cd1f0 https://doi.org/10.1158/2767-9764.29607288

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure S2 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
2Supplementary Figure S3 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
3Supplementary Figure S1 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
4Supplementary Figure S5 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
5Figure 3 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025