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July 21, 2025

Supplementary Figure S3 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer

Ya-Yun Cheng Beth L. Worley Zaineb Javed Amal T. Elhaw Priscilla W. Tang Sarah Al-Saad Shriya Kamlapurkar Sierra R. White Apoorva Uboveja Karthikeyan Mythreye Katherine M. Aird Traci A. Czyzyk Nadine Hempel

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Key Points

MAIN FINDING: Loss of MPZL3 promotes epithelial-mesenchymal transition in ovarian cancer cells.
KEY EVIDENCE: MPZL3 knock-down significantly decreases cell proliferation and disrupts cell cycle progression.
APPROACH: The study evaluated the effects of MPZL3 knock-down on cell dynamics in ovarian cancer.
SIGNIFICANCE: Understanding MPZL3's role may offer insights into ovarian cancer progression and treatment strategies.

Implication

MPZL3 knock-down reduces proliferation and cell cycle progression in ovarian cancer cells, suggesting its role in EMT.

Abstract

Supplementary Figure S3. MPZL3 knock-down decreases proliferation and cell cycle progression.

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Cite This Study

Cheng et al. (2025) studied this question.

synapsesocial.com/papers/689a060ee6551bb0af8cd1ed https://doi.org/10.1158/2767-9764.29607297

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Keep reading

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure S2 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
2Supplementary Figure S1 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
3Supplementary Figure S6 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
4Supplementary Figure S5 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025
5Supplementary Figure S4 from Loss of Predicted Cell Adhesion Molecule MPZL3 Promotes EMT in Ovarian Cancer2025