Ischemic preconditioning protects against myocardial infarction through the activation of A1 adenosine receptors.
Animal data implicate A1 receptors in preconditioning; leaves open translation to clinical cardioprotection.
We conclude that adenosine released during the preconditioning occlusion stimulates cardiac A1 receptors, which leaves the heart protected against infarction even after the adenosine has been withdrawn.
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Liu et al. (1991) studied this question.
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