Key result
The authors propose the hypothesis that genetic expression in shear stress-stimulated endothelial cells is regulated by selective induction of fos and jun gene products.
Population
Endothelial cells
Design
Preclinical
Authors
Loading...
Hypothesis-generating for endothelial mechanotransduction; leaves open fos/jun roles in human vascular disease.
The paper proposes a hypothesis that mechanical stress-induced genetic expression in endothelial cells is mediated by fos and jun gene products.
Nollert et al. (1992) conducted a review in Shear stress-stimulated endothelial cells. Shear stress was evaluated on Genetic expression (fos and jun gene products). The authors propose the hypothesis that genetic expression in shear stress-stimulated endothelial cells is regulated by selective induction of fos and jun gene products.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: