Key result
Stretch-induced increase in intracellular calcium in guinea-pig ventricular myocytes is modulated by extracellular calcium sources and sensitive to verapamil, but not ryanodine or TTX.
Population
Isolated guinea-pig ventricular myocytes loaded with the fluorescent calcium indicator Indo-1
Comparison
Mechanical stretch using carbon fibres, with and… vs Unstretched state or absence of pharmacological…
Design
Preclinical
Authors
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Identifies streptomycin-sensitive calcium entry in stretched myocytes; leaves open relevance to human diastolic dysfunction or arrhythmias.
Stretch-induced increases in resting intracellular calcium in ventricular myocytes are driven by extracellular calcium influx via stretch-activated and calcium-window currents, rather than release from intracellular stores.
Gannier et al. (1996) studied this question. Stretch vs. Baseline / Unstretched state was evaluated on Stretch-induced increase in internal calcium activity ([Ca2+]i). Stretch-induced increase in intracellular calcium in guinea-pig ventricular myocytes is modulated by extracellular calcium sources and sensitive to verapamil, but not ryanodine or TTX.
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