Why the study?
How do high-, low-, and non-nicotine cigarettes affect blood pressure, heart rate, and carbon monoxide levels in smokers with angina pectoris?
How do high-, low-, and non-nicotine cigarettes affect blood pressure, heart rate, and carbon monoxide levels in smokers with angina pectoris?
Nicotine content drives the acute hemodynamic effects of smoking, whereas carbon monoxide exposure occurs regardless of nicotine content.
Answers are grounded in this paper and cite their sources.
Nicotine-dependent hemodynamic effects warrant caution in angina patients; leaves open outcome impact of non-nicotine cigarettes.
Ten cigarette smokers with angina pectoris had blood pressure, heart rate, and expired-air carbon monoxide measurements before and after smoking each of five high-, low-, and non-nicotine cigarettes. There was a significant increase in systolic and diastolic blood pressure after smoking each high- and low-nicotine cigarette, with a significant increase in peak systolic and diastolic blood pressure from cigarette 1 to cigarette 5. There was a significant increase in heart rate after smoking each high- and low-nicotine cigarette but no significant increase in peak heart rate from cigarette 1 to cigarette 5. There was no significant increase in blood pressure or heart rate after smoking a non-nicotine cigarette. There was a significant increase in carbon monoxide level after smoking each high-, low-, and non-nicotine cigarette, with a significant increase in peak carbon monoxide level from cigarette 1 to cigarette 5.
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WlLBERT S. ARONOW (1971) studied this question.
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