In guinea-pig isolated hearts, A2A receptor-mediated coronary vasodilation by adenosine agonists is independent of the endothelium and nitric oxide, but partially dependent on cyclooxygenase products.
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A2A-mediated coronary vasodilation appears NO-independent in guinea-pig hearts; leaves open translation to human in vivo settings.
Maddock et al. (2002) studied this question.
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