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July 1, 2002Journal of Hypertension

Growth characteristics, angiotensin II generation, and microarray-determined gene expression in vascular smooth muscle cells from young spontaneously hypertensive rats

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Authors

WHWen‐Yang HuEast China University of Technology NFNoboru FukudaUniversity of Pittsburgh KKKatsuo KanmatsuseNihon University

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Implication

SHR VSMC abnormalities appear genetically driven; leaves open translation to human hypertension pathogenesis or therapy.

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Cite This Study

Hu et al. (2002) studied this question.

synapsesocial.com/papers/6a853ea1f14ace264a43d578 https://doi.org/10.1097/00004872-200207000-00019

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Molecular Mechanisms of the Exaggerated Growth of Vascular Smooth Muscle Cells in Hypertension1997 · 35 citations
2Contribution of synthetic phenotype on the enhanced angiotensin II-generating system in vascular smooth muscle cells from spontaneously hypertensive rats1999 · 51 citations
3Vascular smooth muscle growth in genetic hypertension1992 · 32 citations
4Role of Endogenous Angiotensin II in the Increased Expression of Growth Factors in Vascular Smooth Muscle Cells from Spontaneously Hypertensive Rats2001 · 50 citations
5Production of Angiotensin II by Homogeneous Cultures of Vascular Smooth Muscle Cells From Spontaneously Hypertensive Rats1999 · 81 citations