September 1, 1989Journal of Hypertension
Molecular biology and pathophysiology of the intrarenal renin-angiotensin system
Population
Experimental heart failure models and spontaneously hypertensive rats (SHR)
Design
Review
Authors
Discussion
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Implication
Altered intrarenal RAS may contribute to HF and hypertension pathophysiology; leaves open whether selective modulation improves outcomes.
Structured PICO
P
Population
Experimental heart failure models and spontaneously hypertensive rats (SHR)
The intrarenal renin-angiotensin system is independently regulated and its altered expression may contribute to the pathophysiology of heart failure and hypertension.
Cite This Study
Dzau et al. (1989) studied this question.