Population
Rabbits made hypertensive by partial constriction of the abdominal aorta
Design
Preclinical
Authors
Loading...
No immediate clinical implications from rabbit vessel studies; leaves open translation of sympathetic hypercontractility mechanisms to human hypertension.
In hypertensive rabbits, increased vascular contractile responses to sympathetic stimulation are primarily driven by increased effector cell mass and neuronal plexus density rather than transmitter disposition changes.
Bevan et al. (2008) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: