Population
Vascular smooth muscle cells from human saphenous vein
Comparison
Angiotensin II vs Serum-free control
Design
Preclinical
Authors
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No immediate clinical implications; leaves open whether AT1-driven VSMC proliferation contributes to human vascular disease.
Angiotensin II acts predominantly via AT1 receptors to induce c-fos expression and modestly increase DNA synthesis in human saphenous vein vascular smooth muscle cells.
Patel et al. (1996) studied this question.
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