Why the study?
Does NG-nitro-L-arginine (NO2Arg) increase blood pressure and inhibit endothelium-dependent relaxation in stroke-prone spontaneously hypertensive rats compared to normotensive rats?
Population
Female stroke-prone spontaneously hypertensive rats (SHRSP) and normotensive Wistar-Kyoto rats (WKY)
Comparison
NG-nitro-L-arginine administered in food for 1… vs Control rats
Design
Preclinical
Follow-up
1 week
Authors
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NO contributes to BP regulation despite hypertension in rats; leaves open translation to human endothelial therapies.
Does NG-nitro-L-arginine (NO2Arg) increase blood pressure and inhibit endothelium-dependent relaxation in stroke-prone spontaneously hypertensive rats compared to normotensive rats?
Endothelium-dependent relaxation contributes to blood pressure regulation in the hypertensive state, despite a baseline decline in this relaxation in stroke-prone spontaneously hypertensive rats.
Kobayashi et al. (1991) studied this question.
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